The discussion of the Linus Pauling vitamin C/lysine invention for chronic scurvy
- Ascorbate Wizard
- Posts: 12878
- Joined: Tue Nov 22, 2005 3:16 pm
- Location: Lisle, IL
The only thing we know from experimental science is that Lp(a) becomes elevated in the absence of sufficient vitamin C. I don't know of any experiments that show Lp(a) decreases after sufficient vitamin C is introduced. There is the early "Reversibility" experiments by Willis, starting with https://vitamincfoundation.org/pdfs/WillisAthero106.pdf that showed in guinea pigs, that CVD can be reversed by introducing vitamin C back into the guinea pig's diet. This implies a reduction in apo(a) (lp(a)). We have the New York Medical professor's experience (after adding proline) and our anecdotal evidence from this forum.
Does the Lp(a) test include the particle size? Small Lp(a) are more atherogenic (dangerous) than larger molecules. And if you Lp(a) is remaining elevated, all the more reason to keep taking the Pauling/Rath Lp(a) binding inhibitors, e.g. vit C, lysine and proline.
Owen R. Fonorow, Follow #OWENRFONOROW at twitter
- Vitamin C Master
- Posts: 241
- Joined: Tue Jan 31, 2017 5:23 am
Perhaps it‘s proline. I‘d like to know how much proline you think I should take. I had my proline value measured 6 months ago. It was 212 nmol/ml and in the middle of the reference range (97-330).
I hope you‘ll give me an answer. Thanks.
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